The paper gives an account of seven cases of inflamatory gastric lesions with infiltration of esinophilus leucocites. Four cases of nodular foros ( gastric inflamatoryfibrous poli pus") and three difuse forms ( "hypertrophic gastroduodemtis with eosinophilus infiltration"). These forms are considered as differing completely from one another and as _having no connection with the so called eosmoph1ll us granuloma of the bone. In gastric inflamatory fibrous polipus, granulation tissue pathogenics is consideredprobable in the case of injuries of the gastric wall with -evolution towards progressive colagenous maturation. The presence of eosinophyllis is accidental, additional and not part of an allergic process. In diffuse foros it is not possible to determine the pathogeny. There is a possibility of a neurohumoral disorder, with excessive permanent stim_uli of the gastric muscles and consecutive parietal hypertrophy, which is more intense in the pilloric ring. There is no proof of an allergic feeding mechanism in these forms.

Se presentan siete casos de lesiones inflamatorias gástricas con infiltración de l!ucocitos eosinófilos. Se relatan cuatro casos de formas nodulares ( "pólipo fibroso inflamatorio gástrico") y tres de formas difusas ( "gastroduodenitis hipertrófica con infiltración eosinófila"). Se consideran formas completamente diferentes entre sí y sin ninguna vinculacióncon el llamado granuloma eosinófilo del hueso. En el pólipo fibroso inflamatorio gástrico se considera como patogenia probable la organización del tejido de gran1lación frente a una injuria de la pared gastnca, que evoluciona a la maduración col